Gestational Age-Dependent Associations Between Mycoplasma/Ureaplasma Colonization and Inflammatory Placental Lesions in Preterm Birth


ÇETİNKAYA DEMİR B., Zadran S., TÜZEMEN N. Ü., KABUL S., ÖZKAN H., ÖZAKIN C.

Journal of Clinical Medicine, cilt.15, sa.10, 2026 (SCI-Expanded, Scopus)

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 15 Sayı: 10
  • Basım Tarihi: 2026
  • Doi Numarası: 10.3390/jcm15103868
  • Dergi Adı: Journal of Clinical Medicine
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus, Chemical Abstracts Core, EMBASE, Academic Search Ultimate (EBSCO), Health Research Premium Collection (ProQuest)
  • Anahtar Kelimeler: Mycoplasma hominis, placental histopathological chorioamnionitis, preterm birth, Ureaplasma urealyticum
  • Bursa Uludağ Üniversitesi Adresli: Evet

Özet

Background: Infection and inflammation are key contributors to spontaneous preterm birth (PTB), but the relationship between genitourinary microbial colonization and placental inflammatory pathology across preterm subgroups remains unclear. Methods: In this case–control study, women with PTB were compared with gestational age-matched controls. Urine cultures, Mycoplasma/Ureaplasma screening, inflammatory markers, and placental histopathology were analyzed. Early (24–33 weeks) and late (34–36 weeks) preterm births were evaluated separately. Results: Clinical risk factors were more common in PTB cases (87.0% vs. 68.7%, p = 0.001), particularly PPROM and fetal growth restriction. Conventional urine culture positivity did not differ between groups. Mycoplasma/Ureaplasma colonization was more frequent in controls (41.2% vs. 15.4%, p < 0.001). Early PTB was strongly associated with placental inflammation, including higher rates of histological chorioamnionitis, composite inflammatory lesions, placental culture positivity, and elevated CRP compared with late PTB. Conclusions: Early PTB may represent a distinct infection-associated phenotype characterized by prominent placental inflammation, whereas late PTB demonstrates a weaker inflammatory profile.